The cellular modifier MOAG‐4/SERF drives amyloid formation through charge complementation | The EMBO Journal
C. elegans maximum velocity correlates with healthspan and is maintained in worms with an insulin receptor mutation | Nature Communications
Frontiers | Comparative Studies in the A30P and A53T α-Synuclein C. elegans Strains to Investigate the Molecular Origins of Parkinson's Disease | Cell and Developmental Biology
Polyglutamine protein aggregates are dynamic | Nature Cell Biology
Protective role of DNJ-27/ERdj5 in Caenorhabditis elegans models of human neurodegenerative diseases. - Abstract - Europe PMC
Protective role of DNJ-27/ERdj5 in Caenorhabditis elegans models of human neurodegenerative diseases. - Abstract - Europe PMC
Molecular chaperones and Parkinson's disease - ScienceDirect
Probiotic Bacillus subtilis Protects against α-Synuclein Aggregation in C. elegans - ScienceDirect
Frontiers | Cellular Regulation of Amyloid Formation in Aging and Disease | Neuroscience
Frontiers | In vivo Pooled Screening: A Scalable Tool to Study the Complexity of Aging and Age-Related Disease | Aging
Bag1–Hsp70 mediates a physiological stress signalling pathway that regulates Raf-1/ERK and cell growth | Nature Cell Biology
Frontiers | Membrane Interactions and Toxicity by Misfolded Protein Oligomers | Cell and Developmental Biology
The cellular modifier MOAG‐4/SERF drives amyloid formation through charge complementation | The EMBO Journal
Frontiers | A Novel Hybrid Model for Drawing Trace Reconstruction from Multichannel Surface Electromyographic Activity | Neuroscience
Genome-wide RNA interference screen identifies previously undescribed regulators of polyglutamine aggregation | PNAS
Age‐ and disease‐specific changes of the kynurenine pathway in Parkinson's and Alzheimer's disease - Sorgdrager - 2019 - Journal of Neurochemistry - Wiley Online Library
The cellular modifier MOAG‐4/SERF drives amyloid formation through charge complementation | The EMBO Journal
Kinetic analysis reveals that independent nucleation events determine the progression of polyglutamine aggregation in C. elegans | PNAS
Identification of an evolutionary conserved structural loop that is required for the enzymatic and biological function of tryptophan 2,3-dioxygenase | Scientific Reports
Protective role of DNJ-27/ERdj5 in Caenorhabditis elegans models of human neurodegenerative diseases. - Abstract - Europe PMC
Protective role of DNJ-27/ERdj5 in Caenorhabditis elegans models of human neurodegenerative diseases. - Abstract - Europe PMC
Identification of MOAG-4/SERF as a Regulator of Age-Related Proteotoxicity - ScienceDirect
Frontiers | Regulation of Age-Related Protein Toxicity | Cell and Developmental Biology
The cellular modifier MOAG‐4/SERF drives amyloid formation through charge complementation | The EMBO Journal
